Melatonin Biomarkers and Delirium in Older Adults: A Systematic Review of Observational Studies​

Abstract ID
5424
Authors' names
Ganre Akpubi1, Michaela Gilarova2, Szu-Chia Huang2, Salomé Andres2, Gillian E Mead3, Tughrul Arslan4,2, Alasdair M J MacLullich3, Renata L Riha5,6, Dr Maria Gardani7​
Author's provenances
1. Edinburgh Medical School, University of Edinburgh, Edinburgh, United Kingdom; 2. Advanced Care Research Centre, Usher Institute, University of Edinburgh, Edinburgh, United Kingdom; 3. Centre for Population Health Sciences, Usher Institute, University o
Abstract category
Conditions

Abstract

Melatonin biomarkers and delirium in older adults: A systematic review of observational studies 

Introduction 

Delirium, a severe clinical syndrome characterised by acute disturbances in attention, awareness and cognition, affects approximately 20–50% of older adults in hospital care. Melatonin, a key regulator of circadian rhythms, has been proposed as a potential biomarker for delirium due to its role in sleep–wake regulation. However, evidence regarding the association between melatonin secretion and delirium remains inconsistent. This systematic review synthesised evidence on the association between melatonin biomarkers and delirium in older adults. 

Methods 

Six databases (MEDLINE, Embase, PsycINFO, CINAHL Plus, Scopus, and Web of Science) were systematically searched up to 1 June 2026 to identify observational studies assessing melatonin biomarkers and delirium in older adults (aged ≥60 years). Findings were synthesised narratively. 

Results 

Eleven observational studies involving 1,782 participants were included (32.0% with delirium). Eight studies measured melatonin concentrations in biological samples (serum, plasma, saliva, urine and cerebrospinal fluid), while three investigated genetic variants of the melatonin receptor gene MTNR1B. Most were prospective cohort studies conducted in surgical or intensive care settings. Six of the eight biomarker studies reported altered melatonin secretion or disrupted circadian rhythms in patients with delirium, although findings were heterogeneous, with several studies reporting lower melatonin concentrations while others found no association or higher postoperative levels. Genetic findings were limited and inconsistent. Heterogeneity in study populations, biological sample type, timing of melatonin measurement and delirium assessment methods limited direct comparison across studies or meta-analyses.  

Conclusion 

Current evidence suggests that altered melatonin regulation may be associated with delirium in older adults. However, methodological heterogeneity and inconsistent findings limit firm conclusions. Further large, prospective studies using standardised melatonin measurement and delirium assessment are needed to determine the utility of melatonin as a biomarker for delirium.